Service for patients: Call us right away at +43 2622 39 501!
Please feel free to call us: +43 2622 39 501
ordination@lissimed.at
Phone
+43 2622 39 501
Ordination
Eyerspergring 6-8, 2700 Wr. Neustadt
ordination@lissimed.at
+43 2622 39 501
Eyerspergring 6-8, 2700 Wr. Neustadt
Helpful, state-of-the-art medicine is not possible without special consideration of environmental medicine.
Chelation therapy is an important component of this.
Highly toxic metals such as mercury, lead, arsenic, cadmium, tin etc. can be detected in almost all patients.
The special thing about this is that these metals are not only found in the blood, but are deposited in all tissues of the body.
As they are particularly fat-soluble (lipophilic), they particularly like to be deposited in nerve tissue, which is extremely rich in fat, and form deposits there
The individual detoxification capacity of people varies greatly.
In chronically ill people, we generally find a limited and impaired detoxification capacity.
We can measure this very precisely via the enzymes of glutathione S-transferase (GST T1, GST M1, GST P1) as well as via NAT 2 and SOD2 (phase II) and via individually selected phase I enzymes (see environmental toxin diagnostics)
With DMPS, DMSA and EDTA infusions, we can selectively wash highly toxic metals out of the nerve tissue, connective tissue and internal organs.
The majority of our patients report significant improvements in their state of health.
Metal exposure can play a causal role in the development of chronic inflammatory multisystem diseases by directly promoting inflammation and at the same time negatively influencing the regulatory cycle between inflammation, mitochondriopathy, oxidative and nitrosative stress.
In this respect, they play a decisive role in permanently disrupting immune tolerance through chronic inflammation, which in turn makes the organism more “sensitive” and intolerant to numerous other trigger factors.
This connection explains the “breadth” of diseases associated with metal effects.
See also the Cercla list.
Metal exposure can play a role in numerous common diseases
Studies suggest that metal-induced oxidative stress may not only be significantly involved in the pathogenesis of arteriosclerosis, but also in the pathogenesis of type 2 diabetes. For example, a reduction in copper exposure using chelation therapy lowers both the production of free radicals and insulin resistance in the mouse model for diabetes.
Lead exposure leads to oxidative damage to blood vessels and thus promotes the progression of chronic obstructive pulmonary disease and chronic renal insufficiency.
The pro-inflammatory effect of metals can also be clinically prominent.
By releasing cobalt wear particles from hip implants, cobalt activates the Toll-like receptor 4 (TLR4) signaling pathway and increases the release of TNF-alpha and IL-8. In fact, cobalt blood levels correlate with the likelihood of requiring revision of the artificial joint.
Chelation therapy has always been particularly important in the treatment of arteriosclerosis.
The success of this treatment can usually be precisely monitored sonographically (using ultrasound).
Individual sensitivity/receptivity is important
In addition to the extent of the metal exposure, the individual sensitivity/susceptibility of the patient is always important for the type and extent of the symptoms.
This concerns a certain genetically determined sensitivity (e.g. phase II detoxification by GST enzymes etc.), but above all the functional resistance, i.e. the ability of the organism to compensate for and repair any stress and damage that occurs.
For example, a good supply of essential trace elements (recognizable in the whole blood mineral profile), a sufficient supply of vitamins (B and D vitamins, folic acid, coenzyme Q10) and antioxidants promote “resistance”.
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